LENTIVIRUS — this is a definition from Stanford University’s Lentivirus Fact Sheet. A Lentivirus produces “multi-organ diseases characterized by long incubation periods and persistent infection.”
Wikipedia defines the Lentivirus this way:
Lentivirus is a genus of retroviruses that cause chronic and deadly diseases characterized by long incubation periods, in humans and other mammalian species. The genus includes the human immunodeficiency virus (HIV), which causes AIDS. Lentiviruses are distributed worldwide, and are known to be hosted in apes, cows, goats, horses, cats, and sheep as well as several other mammals.
Lentiviruses can integrate a significant amount of viral complementary DNA into the DNA of the host cell and can efficiently infect nondividing cells, so they are one of the most efficient methods of gene delivery. They can become endogenous, integrating their genome into the host germline genome, so that the virus is henceforth inherited by the host’s descendants.
Chronic and deadly diseases that have long incubation periods, such as HIV. They integrate into the DNA and are passed on to the host’s descendants. This is alarming.
What are the hazards of Lentiviruses? From Stanford University —
In terms of the pathogenesis of lentivirus, some key properties are:
- Lentiviruses persist lifelong. This is a function both of their ability to integrate into the host chromosome and of their ability to evade host immunity. This ability to evade host immunity may be related both to the high mutation rates of these viruses, and to their ability to infect immune cells (macrophages, and in the case of HIV, T-cells).
- Lentiviruses have high mutation rates. Lentiviruses replicate, mutate and undergo selection by host immune responses.
- Infection proceeds through at least three stages.
- Initial (acute) lentivirus infection is associated with rapid viral replication and dissemination, which is often accompanied by a transient period of disease.
- This is followed by a latent period, during which the virus is brought under immune control and no disease occurs.
- High levels of viral replication then resume at some later time, leading to disease.
Acute infection with human lentiviruses can appear as non-specific “flu-like” and “mononucleosislike” symptoms, including myalgia, arthralgia, diarrhea, nausea, vomiting, headache, hepatosplenomegaly, weight loss and neurological symptoms.
Transmitted from person to person through direct exposure to infected body fluids (blood, semen) sexual contact, sharing unclean needles etc.; transplacental transfer can occur.
Direct contact with skin and mucous membranes of the eye, nose and mouth; accidental parenteral injection; ingestion; hazard of aerosols exposure unknown; insertional mutagenesis; integration and expression of oncogenes or potential oncogenes.
Lentiviruses persist life long — you can’t get rid of them. They have high mutation rates, evading the immune response, and infect other cells. After the initial acute infection they have a long latency period before they resume replication and cause disease. They can be transmitted by bodily fluids, and they can be passed on to descendants.
They are used in the COVID “vaccines”.